| 16516385 |
15516 |
Hsp90ab1
|
Oxidative Stress |
We verified array results with RT-PCR and Northern blotting for three genes which are related to oxidative damage closely, including Hsp84, Hsp86 and YB-1. |
| 16516385 |
15519 |
Hsp90aa1
|
Oxidative Stress |
We verified array results with RT-PCR and Northern blotting for three genes which are related to oxidative damage closely, including Hsp84, Hsp86 and YB-1. |
| 16516385 |
22608 |
Ybx1
|
Oxidative Stress |
We verified array results with RT-PCR and Northern blotting for three genes which are related to oxidative damage closely, including Hsp84, Hsp86 and YB-1. |
| 25983633 |
216343 |
Tph2
|
Oxidative Stress |
Therefore, the oxidative processes that prevail in PD may cause a misfolding of Tph2 and result in the modification of serotonin function such as is seen in dopamine neurons. |
| 26016857 |
18024 |
Nfe2l2
|
Oxidative Stress |
It is well known that Nrf2 suppresses neurodegeneration via a mechanism involving the inhibition of oxidative stress and gliosis. |
| 31412703 |
15516 |
Hsp90ab1
|
Oxidative Stress |
The levels of oxidative stress and protein carbonyl, mRNA and protein expression levels of Hsp84 and Hsp86 in the hippocampus were detected. |
| 31412703 |
15516 |
Hsp90ab1
|
Oxidative Stress |
CONCLUSION: Acupuncture may delay brain ageing in SAMP8 mice by reducing oxidative protein damage and promoting Hsp84 and Hsp86 expression. |
| 31412703 |
15519 |
Hsp90aa1
|
Oxidative Stress |
The levels of oxidative stress and protein carbonyl, mRNA and protein expression levels of Hsp84 and Hsp86 in the hippocampus were detected. |
| 31412703 |
15519 |
Hsp90aa1
|
Oxidative Stress |
CONCLUSION: Acupuncture may delay brain ageing in SAMP8 mice by reducing oxidative protein damage and promoting Hsp84 and Hsp86 expression. |
| 33341053 |
232975 |
Atp1a3
|
Oxidative Stress |
Taken together, our data suggested that acupuncture was effective in treating asthma by attenuation of AHR, mucus secretion and airway inflammation, and the mechanism was associated with regulation of ferroptosis, SLC3A2 and ATP1A3 protein expression as well as oxidative stress. |
| 33341053 |
17254 |
Slc3a2
|
Oxidative Stress |
Taken together, our data suggested that acupuncture was effective in treating asthma by attenuation of AHR, mucus secretion and airway inflammation, and the mechanism was associated with regulation of ferroptosis, SLC3A2 and ATP1A3 protein expression as well as oxidative stress. |
| 22964493 |
24482 |
Igf1
|
Oxidative Stress |
CONCLUSION: These findings suggest that the anti-hypertensive mechanism of EA could be related to the effects of oxidative stress on IGF-I in RF-induced hypertension. |
| 22964493 |
24482 |
Igf1
|
Oxidative Stress |
The effect of electroacupuncture on insulin-like growth factor-I and oxidative stress in an animal model of renal failure-induced hypertension. |
| 25302705 |
245965 |
Atp5f1d
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
29754 |
Atp5mc1
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
171082 |
Atp5mc2
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
114630 |
Atp5mc3
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
300677 |
Atp5mg
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
25414 |
Bcar1
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
29161 |
Cav3
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
29393 |
Col1a1
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
84352 |
Col1a2
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
85490 |
Col5a1
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
60379 |
Col5a3
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
361821 |
Col6a2
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
94194 |
Cox5b
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
300047 |
Cyc1
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
362332 |
Flnc
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
140433 |
Lama5
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
296313 |
Myl9
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
299643 |
Ndufa7
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
362344 |
Ndufb2
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
361385 |
Ndufb7
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
293991 |
Ndufb8
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
362837 |
Ndufs7
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
293652 |
Ndufs8
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
25105 |
Nppb
|
Oxidative Stress |
We also found that electro-acupuncture repressed Nppb expression (GSE61840), suggesting its protection against oxidative stress. |
| 25302705 |
24842 |
Tp53
|
Oxidative Stress |
In addition, some pathways were uniquely regulated by EA, but not NA pretreatment, such as oxidative stress, cardiac muscle contraction, gap junction, vascular smooth muscle contraction, hypertrophic, NOD-like receptor, and P53 and B-cell receptor pathways. |
| 25302705 |
94203 |
Pgf
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
295342 |
Rhoc
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 25302705 |
89811 |
Vegfb
|
Oxidative Stress |
On the other hand, the down-regulated genes are mainly involved in oxidative phosphorylation (e.g., Atp5d, Ndufb7, Ndufb8, Ndufa7, Cyc1, Atp5g2, Atp5g1, Cox5b, Atp5g3, Ndufb2, Ndufs7, Ndufs8, Cos6a1, Atp5l) and focal adhesion (e.g., Cav3, Pgf, Bcar1, Col5a3, Flnc, Col5a1, Myl9, Vegfb, Lama5, Col6a2, Col1a2, Rhoc, Col1a1) pathways. |
| 26276454 |
24248 |
Cat
|
Oxidative Stress |
At the end of study, brain oxidative status including malondialdehyde levels and the activities of superoxide dismutase, catalase, and glutathione peroxidase were determined in the cortex, striatum, and hippocampus. |
| 28478802 |
66021 |
Cybb
|
Oxidative Stress |
Electroacupuncture ameliorates spatial learning and memory impairment via attenuating NOX2-related oxidative stress in a rat model of Alzheimer's disease induced by Abeta1-42. |
| 28478802 |
66021 |
Cybb
|
Oxidative Stress |
The present study aimed to determine the effects of EA treatment on spatial learning and memory impairment, and to elucidate the status of NOX2-related oxidative stress in a rat model of Alzheimer's disease induced by Beta-amyloid1-42 (Abeta1-42). |
| 28478802 |
66021 |
Cybb
|
Oxidative Stress |
Our findings suggested that EA is a potential strategy for the treatment of AD, and the possible mechanism is associated with the alleviation of neuronal injury and inhibition of NOX2-related oxidative stress. |
| 28493083 |
25402 |
Casp3
|
Oxidative Stress |
EAc reversed the CPB-induced lung inflammation, oxidative damage, and apoptosis; the mechanism may involve decreased phosphorylation of p38 along with caspase-3 activity and activation. |
| 28493083 |
81649 |
Mapk14
|
Oxidative Stress |
EAc reversed the CPB-induced lung inflammation, oxidative damage, and apoptosis; the mechanism may involve decreased phosphorylation of p38 along with caspase-3 activity and activation. |
| 28889839 |
81632 |
Abat
|
Oxidative Stress |
At the end of study, the changes of oxidative stress markers, the expressions of cytokines interleukin 6 (IL-6) and glutamic acid decarboxylase (GAD) proteins (65 kDa and 67 kDa) together with gamma-aminobutyric acid transaminase (GABA-T) activity and density of Purkinje cell in the cerebellum were assessed. |
| 28889839 |
81632 |
Abat
|
Oxidative Stress |
The results showed that laser acupuncture HT7 decreased oxidative stress, IL-6 expression, and GABA-T activity but increased the expressions of GAD 65 kDa together with the density of Purkinje cells in the cerebellum. |
| 28889839 |
25246 |
Bsg
|
Oxidative Stress |
Based on the benefit of laser acupuncture at HT7 acupoint on the neurological disorders related with oxidative stress and inflammation, its benefit on oxidative stress, neuroinflammation, and GABAergic/glutamatergic imbalance in cerebellum of autism have been considered. |
| 28889839 |
25246 |
Bsg
|
Oxidative Stress |
The results showed that laser acupuncture HT7 decreased oxidative stress, IL-6 expression, and GABA-T activity but increased the expressions of GAD 65 kDa together with the density of Purkinje cells in the cerebellum. |
| 28889839 |
24498 |
Il6
|
Oxidative Stress |
At the end of study, the changes of oxidative stress markers, the expressions of cytokines interleukin 6 (IL-6) and glutamic acid decarboxylase (GAD) proteins (65 kDa and 67 kDa) together with gamma-aminobutyric acid transaminase (GABA-T) activity and density of Purkinje cell in the cerebellum were assessed. |
| 28889839 |
24498 |
Il6
|
Oxidative Stress |
The results showed that laser acupuncture HT7 decreased oxidative stress, IL-6 expression, and GABA-T activity but increased the expressions of GAD 65 kDa together with the density of Purkinje cells in the cerebellum. |
| 30431067 |
24599 |
Nos2
|
Oxidative Stress |
The production of pro-oxidative stress factors, including nitric oxide (NO) and inducible nitric oxide synthase (iNOS), was also blocked along with the amelioration of cognitive function, while the production of adenosine triphosphate (ATP), superoxide dismutase (SOD) and cyclooxygenase (COX) was restored. |
| 30431067 |
24786 |
Sod1
|
Oxidative Stress |
The production of pro-oxidative stress factors, including nitric oxide (NO) and inducible nitric oxide synthase (iNOS), was also blocked along with the amelioration of cognitive function, while the production of adenosine triphosphate (ATP), superoxide dismutase (SOD) and cyclooxygenase (COX) was restored. |
| 30431067 |
24693 |
Ptgs1
|
Oxidative Stress |
The production of pro-oxidative stress factors, including nitric oxide (NO) and inducible nitric oxide synthase (iNOS), was also blocked along with the amelioration of cognitive function, while the production of adenosine triphosphate (ATP), superoxide dismutase (SOD) and cyclooxygenase (COX) was restored. |
| 35405257 |
24225 |
Bdnf
|
Oxidative Stress |
MATERIALS AND METHODS: Adopted middle-cerebral artery occlusion/reperfusion (MCAO/R) rat models, the effect for the stroke of the combination treatment consisting of acupuncture and NMT was evaluated by detecting neurological issues, cerebral infarct dimensions, levels of inflammatory factors (IL-6, IL-1beta, TNF-alpha) and oxidative stress factors (SOD, MDA) and brain-derived neurotrophic factor (BDNF). |
| 35405257 |
24494 |
Il1b
|
Oxidative Stress |
MATERIALS AND METHODS: Adopted middle-cerebral artery occlusion/reperfusion (MCAO/R) rat models, the effect for the stroke of the combination treatment consisting of acupuncture and NMT was evaluated by detecting neurological issues, cerebral infarct dimensions, levels of inflammatory factors (IL-6, IL-1beta, TNF-alpha) and oxidative stress factors (SOD, MDA) and brain-derived neurotrophic factor (BDNF). |
| 35405257 |
24498 |
Il6
|
Oxidative Stress |
MATERIALS AND METHODS: Adopted middle-cerebral artery occlusion/reperfusion (MCAO/R) rat models, the effect for the stroke of the combination treatment consisting of acupuncture and NMT was evaluated by detecting neurological issues, cerebral infarct dimensions, levels of inflammatory factors (IL-6, IL-1beta, TNF-alpha) and oxidative stress factors (SOD, MDA) and brain-derived neurotrophic factor (BDNF). |
| 35405257 |
24835 |
Tnf
|
Oxidative Stress |
MATERIALS AND METHODS: Adopted middle-cerebral artery occlusion/reperfusion (MCAO/R) rat models, the effect for the stroke of the combination treatment consisting of acupuncture and NMT was evaluated by detecting neurological issues, cerebral infarct dimensions, levels of inflammatory factors (IL-6, IL-1beta, TNF-alpha) and oxidative stress factors (SOD, MDA) and brain-derived neurotrophic factor (BDNF). |
| 32249904 |
24835 |
Tnf
|
Oxidative Stress |
As shown in Figure 4C, in the warm acupuncture-treated group, when compared with the model groups, the proteins with differential levels were enriched in the KEGG pathways of cardiac muscle contraction, endocytosis, platelet activation, actin cytoskeleton regulation, complement and coagulation cascades, apelin signaling, oxytocin signaling, amino acid biosynthesis, autophagy, axon singling, Alzheimer disease, non-alcoholic fatty liver disease (NAFLD), cytokine-cytokine receptor interaction, spliceosome, chemokine signaling, TNF-alpha signaling, neuroactive ligand-receptor interaction, phagocytosis, oxidative phosphorylation and autoimmune thyroid disease (Supplementary Table S8). |
| 30906419 |
24932 |
Cd4
|
Oxidative Stress |
In addition, coexpressed DEGs networks indicated that Cd4 and Il-33 might mediate the cascade of inflammation and oxidative stress responses, which could serve as a potential target of acupuncture treatment. |
| 30906419 |
361749 |
Il33
|
Oxidative Stress |
In addition, coexpressed DEGs networks indicated that Cd4 and Il-33 might mediate the cascade of inflammation and oxidative stress responses, which could serve as a potential target of acupuncture treatment. |
| 34405366 |
25319 |
Fth1
|
Oxidative Stress |
Within the context of oxidative stress, Nrf2 is an essential moderator (Kang and Tang ); its vital downstream proteins include GPX4 and FTH1. |
| 34405366 |
29328 |
Gpx4
|
Oxidative Stress |
GPX4 is a key participant in oxidative stress equilibrium (Li et al. |
| 34405366 |
29328 |
Gpx4
|
Oxidative Stress |
In combination, these data infer that SA may diminish the oxidative stress damage precipitated by ICH by amplifying GPX4 expression. |
| 34405366 |
29328 |
Gpx4
|
Oxidative Stress |
Within the context of oxidative stress, Nrf2 is an essential moderator (Kang and Tang ); its vital downstream proteins include GPX4 and FTH1. |
| 34405366 |
83619 |
Nfe2l2
|
Oxidative Stress |
Within the context of oxidative stress, Nrf2 is an essential moderator (Kang and Tang ); its vital downstream proteins include GPX4 and FTH1. |
| 36484920 |
29560 |
Hif1a
|
Oxidative Stress |
Bloodletting Acupuncture at Jing-Well Points Alleviates Myocardial Injury in Acute Altitude Hypoxic Rats by Activating HIF-1alpha/BNIP3 Signaling-Mediated Mitochondrial Autophagy and Decreasing Oxidative Stress. |
| 36484920 |
29560 |
Hif1a
|
Oxidative Stress |
CONCLUSION: BAJP is an effective treatment for AHH-induced myocardial injury, and the mechanism might involve increasing HIF-1alpha/BNIP3 signaling-mediated autophagy and decreasing oxidative stress. |
| 36484920 |
84480 |
Bnip3
|
Oxidative Stress |
Bloodletting Acupuncture at Jing-Well Points Alleviates Myocardial Injury in Acute Altitude Hypoxic Rats by Activating HIF-1alpha/BNIP3 Signaling-Mediated Mitochondrial Autophagy and Decreasing Oxidative Stress. |
| 36484920 |
84480 |
Bnip3
|
Oxidative Stress |
CONCLUSION: BAJP is an effective treatment for AHH-induced myocardial injury, and the mechanism might involve increasing HIF-1alpha/BNIP3 signaling-mediated autophagy and decreasing oxidative stress. |
| 15894008 |
116484 |
Txn1
|
Oxidative Stress |
the thioredoxin (Trx) system (consisting of thioredoxin reductase (TR), Trx and NADPH) prevents susceptible proteins from this oxidative modification. |
