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Basic Characteristics of Mutations
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Mutation Site
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H105P |
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Mutation Site Sentence
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Mutations His105Pro, Leu364Pro and Asp162Ala lead to the loss of TK activity that could result in ACV-resistance. |
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Mutation Level
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Amino acid level |
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Mutation Type
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Nonsynonymous substitution |
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Gene/Protein/Region
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TK |
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Standardized Encoding Gene
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UL23
|
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Genotype/Subtype
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- |
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Viral Reference
|
-
|
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Functional Impact and Mechanisms
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Disease
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Herpes simplex
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Immune
|
- |
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Target Gene
|
-
|
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Clinical and Epidemiological Correlations
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Clinical Information
|
- |
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Treatment
|
ACV |
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Location
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- |
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Literature Information
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PMID
|
16970995
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|
Title
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Resistance of herpes simplex virus type 1 to acyclovir: thymidine kinase gene mutagenesis study
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Author
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Frobert E,Ooka T,Cortay JC,Lina B,Thouvenot D,Morfin F
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Journal
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Antiviral research
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Journal Info
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2007 Feb;73(2):147-50
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Abstract
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By site-directed mutagenesis, we investigate the role of six mutations of herpes simplex virus type 1 thymidine kinase (TK) gene in the acquisition of resistance to acyclovir (ACV). TK activity was not impaired by substitutions located at codons 17, 161 and 374 and these mutations were thus related to TK gene polymorphism. Mutations His105Pro, Leu364Pro and Asp162Ala lead to the loss of TK activity that could result in ACV-resistance.
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Sequence Data
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-
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