|
Basic Characteristics of Mutations
|
|
Mutation Site
|
I2230T |
|
Mutation Site Sentence
|
Our results further showed that at least two site mutations in B.1.1.7 resulted in a decrease in CD8+ T cell activation and a possible immune evasion, namely A1708D mutation in ORF1ab1707-1716 and I2230T mutation in ORF1ab2230-2238. |
|
Mutation Level
|
Amino acid level |
|
Mutation Type
|
Nonsynonymous substitution |
|
Gene/Protein/Region
|
ORF1ab |
|
Standardized Encoding Gene
|
ORF1ab
|
|
Genotype/Subtype
|
B.1.1.7 |
|
Viral Reference
|
-
|
|
Functional Impact and Mechanisms
|
|
Disease
|
COVID-19
|
|
Immune
|
- |
|
Target Gene
|
-
|
|
Clinical and Epidemiological Correlations
|
|
Clinical Information
|
- |
|
Treatment
|
- |
|
Location
|
- |
|
Literature Information
|
|
PMID
|
35194575
|
|
Title
|
SARS-CoV-2 variant B.1.1.7 caused HLA-A2(+) CD8(+) T cell epitope mutations for impaired cellular immune response
|
|
Author
|
Xiao C,Mao L,Wang Z,Gao L,Zhu G,Su J,Chen X,Yuan J,Hu Y,Yin Z,Xie J,Ji W,Niu H,Gao F,Luo OJ,Xiao L,Wang P,Chen G
|
|
Journal
|
iScience
|
|
Journal Info
|
2022 Mar 18;25(3):103934
|
|
Abstract
|
Here, we evaluated the immune properties of the HLA-A2 restricted CD8(+) T cell epitopes containing mutations from B.1.1.7, and furthermore performed a comprehensive analysis of the SARS-CoV-2 specific CD8(+) T cell responses from COVID-19 convalescent patients and SARS-CoV-2 vaccinees recognizing the ancestral Wuhan strain compared to B.1.1.7. First, most of the predicted CD8(+) T cell epitopes showed proper binding with HLA-A2, whereas epitopes from B.1.1.7 had lower binding capability than those from the ancestral strain. In addition, these peptides could effectively induce the activation and cytotoxicity of CD8(+) T cells. Our results further showed that at least two site mutations in B.1.1.7 resulted in a decrease in CD8(+) T cell activation and a possible immune evasion, namely A1708D mutation in ORF1ab(1707-1716) and I2230T mutation in ORF1ab(2230-2238). Our current analysis provides information that contributes to the understanding of SARS-CoV-2-specific CD8(+) T cell responses elicited by infection of mutated strains or vaccination.
|
|
Sequence Data
|
-
|
|
|