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Basic Characteristics of Mutations
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Mutation Site
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K200R |
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Mutation Site Sentence
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By passage 5, the E2-K200R;44-UTR mutant virus was the dominant virus in the cell culture supernatant (p < 0.0001); a similar shift in virus proportions was not observed in the E2-K200R* versus E2-K200R competition (Figure 2A). |
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Mutation Level
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Amino acid level |
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Mutation Type
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Nonsynonymous substitution |
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Gene/Protein/Region
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E2 |
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Standardized Encoding Gene
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E2
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Genotype/Subtype
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- |
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Viral Reference
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AF15561
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Functional Impact and Mechanisms
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Disease
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Chikungunya Fever
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Immune
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- |
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Target Gene
|
-
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Clinical and Epidemiological Correlations
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Clinical Information
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- |
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Treatment
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- |
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Location
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- |
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Literature Information
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PMID
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38932154
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Title
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A 44-Nucleotide Region in the Chikungunya Virus 3' UTR Dictates Viral Fitness in Disparate Host Cells
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Author
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Ander SE,Carpentier KS,Sanders W,Lucas CJ,Jolly AJ,Johnson CN,Hawman DW,Heise MT,Moorman NJ,Morrison TE
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Journal
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Viruses
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Journal Info
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2024 May 28;16(6):861
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Abstract
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We previously reported that deletion of a 44-nucleotide element in the 3' untranslated region (UTR) of the Chikungunya virus (CHIKV) genome enhances the virulence of CHIKV infection in mice. Here, we find that while this 44-nucleotide deletion enhances CHIKV fitness in murine embryonic fibroblasts in a manner independent of the type I interferon response, the same mutation decreases viral fitness in C6/36 mosquito cells. Further, the fitness advantage conferred by the UTR deletion in mammalian cells is maintained in vivo in a mouse model of CHIKV dissemination. Finally, SHAPE-MaP analysis of the CHIKV 3' UTR revealed this 44-nucleotide element forms a distinctive two-stem-loop structure that is ablated in the mutant 3' UTR without altering additional 3' UTR RNA secondary structures.
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Sequence Data
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-
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