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Basic Characteristics of Mutations
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Mutation Site
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Q80K |
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Mutation Site Sentence
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RESULTS: Ten patients (10.5%) had a Q80K mutation and 12 patients exhibited bilirubin levels above the upper limit of normal at baseline. |
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Mutation Level
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Amino acid level |
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Mutation Type
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Nonsynonymous substitution |
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Gene/Protein/Region
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Protease |
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Standardized Encoding Gene
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NS3
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Genotype/Subtype
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1a |
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Viral Reference
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AF009606
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Functional Impact and Mechanisms
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Disease
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HCV Infection
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Immune
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- |
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Target Gene
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-
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Clinical and Epidemiological Correlations
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Clinical Information
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- |
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Treatment
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- |
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Location
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France |
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Literature Information
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PMID
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25027573
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Title
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Patients eligible for treatment with simeprevir in a French center
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Author
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Morel V,Duverlie G,Brochot E
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Journal
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Journal of clinical virology : the official publication of the Pan American Society for Clinical Virology
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Journal Info
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2014 Sep;61(1):149-51
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Abstract
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BACKGROUND: Simeprevir (Olysio((R))), a second-wave protease inhibitor recently approved for the treatment of chronic hepatitis C, is not indicated in patients with genotype 1a strain harboring the Q80K protease mutation. Phase 2 and 3 studies on this molecule mainly focused on North American patients and the prevalence of Q80K is particularly high in the USA (around 50%). The prevalence of this mutation in other parts of the world is currently unknown. OBJECTIVES: The purpose of our study was to perform a detection of this mutation in a single PCR technique and to study the prevalence of this Q80K mutation in a non U.S. population. We can thus estimate the proportion of HCV positive patients who can be treated with simeprevir. STUDY DESIGN: We conducted a meta-analysis of response rates in the presence or absence of this mutation in randomized trials and then describe a simple and reliable method to detect this mutation. We also examined bilirubin levels in our cohort of 95 HCV genotype 1a patients. RESULTS: Ten patients (10.5%) had a Q80K mutation and 12 patients exhibited bilirubin levels above the upper limit of normal at baseline. In our cohort, 21 patients (22%) were therefore ineligible for treatment with simeprevir. The prevalence of this mutation seems to be much lower in European patients. CONCLUSION: In conclusion, before considering treatment with simeprevir, physicians must be able to screen for the Q80K mutation.
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Sequence Data
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-
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