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Basic Characteristics of Mutations
|
|
Mutation Site
|
R292K |
|
Mutation Site Sentence
|
Following treatment with oseltamivir, neuraminidase (NA) gene sequencing indicated the presence of an R292K mutation. |
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Mutation Level
|
Amino acid level |
|
Mutation Type
|
Nonsynonymous substitution |
|
Gene/Protein/Region
|
NA |
|
Standardized Encoding Gene
|
NA
|
|
Genotype/Subtype
|
H3N2 |
|
Viral Reference
|
-
|
|
Functional Impact and Mechanisms
|
|
Disease
|
Influenza A
|
|
Immune
|
Y |
|
Target Gene
|
-
|
|
Clinical and Epidemiological Correlations
|
|
Clinical Information
|
- |
|
Treatment
|
oseltamivir |
|
Location
|
Canada |
|
Literature Information
|
|
PMID
|
27442795
|
|
Title
|
Oseltamivir resistance in an influenza A (H3N2) virus isolated from an immunocompromised patient during the 2014-2015 influenza season in Alberta, Canada
|
|
Author
|
Chaudhry A,Bastien N,Li Y,Scott A,Pabbaraju K,Stewart D,Wong S,Drews SJ
|
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Journal
|
Influenza and other respiratory viruses
|
|
Journal Info
|
2016 Nov;10(6):532-535
|
|
Abstract
|
This manuscript describes the identification of an oseltamivir-resistant influenza A (H3N2) virus in a respiratory specimen collected from an immunocompromised patient in Alberta, Canada, during the 2014-2015 influenza season. Following treatment with oseltamivir, neuraminidase (NA) gene sequencing indicated the presence of an R292K mutation. Phenotypic susceptibility testing by the NA-Star assay indicated a highly reduced inhibition by oseltamivir and normal inhibition by zanamivir. The use of zanamivir following identification of the oseltamivir-resistant strain, combined with a partial immune reconstitution, was followed by a suggested decrease in the nasopharyngeal viral load in the nasopharynx and clinical improvement of the patient.
|
|
Sequence Data
|
-
|