|
Basic Characteristics of Mutations
|
|
Mutation Site
|
V27I |
|
Mutation Site Sentence
|
We found a relatively high frequency of unique amantadine-resistant H3N2 and H9N2 avian influenza viruses (Val27Ile on M2 protein) isolated from live poultry markets in South Korea and confirmed that a Val27Ile single substitution in the M2 protein is enough to acquire the amantadine resistance phenotype by using reverse-genetically created human-avian reassortant viruses. |
|
Mutation Level
|
Amino acid level |
|
Mutation Type
|
Nonsynonymous substitution |
|
Gene/Protein/Region
|
M2 |
|
Standardized Encoding Gene
|
M
|
|
Genotype/Subtype
|
H3N2 |
|
Viral Reference
|
A/duck/Korea/L03/05 wild type
|
|
Functional Impact and Mechanisms
|
|
Disease
|
Influenza A
|
|
Immune
|
- |
|
Target Gene
|
-
|
|
Clinical and Epidemiological Correlations
|
|
Clinical Information
|
- |
|
Treatment
|
amantadine |
|
Location
|
South Korea |
|
Literature Information
|
|
PMID
|
18845822
|
|
Title
|
Emergence of amantadine-resistant H3N2 avian influenza A virus in South Korea
|
|
Author
|
Lee J,Song YJ,Park JH,Lee JH,Baek YH,Song MS,Oh TK,Han HS,Pascua PN,Choi YK
|
|
Journal
|
Journal of clinical microbiology
|
|
Journal Info
|
2008 Nov;46(11):3788-90
|
|
Abstract
|
We found a relatively high frequency of unique amantadine-resistant H3N2 and H9N2 avian influenza viruses (Val27Ile on M2 protein) isolated from live poultry markets in South Korea and confirmed that a Val27Ile single substitution in the M2 protein is enough to acquire the amantadine resistance phenotype by using reverse-genetically created human-avian reassortant viruses.
|
|
Sequence Data
|
-
|